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Общее количество найденных документов : 55
Показаны документы с 1 по 20
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1.
| Transforming growth factor 'бета'[1]: protective effect against experimental autoimmune diseases and inhibition of contact hypersensitivity responses in mice // 11th Eur. Immunol. Meet., Espoo, 9-12 June, 1991. -Helsinki, 1991.-С.36
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2.
| Warfvinge G. Monoclonal antibody reactivity of oral mucosa in experimental contact hypersensitivity // Langerhans Cell.. -Paris, London, 1988.-С.421-429
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3.
| Study of the kinetics of Ia antigen positive dendritic cells in mice contact hypersensitivity reaction // Langerhans Cell. -Paris, London, 1988.-С.397-404
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4.
| Dendritic cells from control but not atopic donors respond to contact and respiratory sensitizer treatment in vitro with differential cytokine production and altered stimulatory capacity // Clin. and Exp. Allergy, 2008. Vol. 38, N 7.-С.1148-1159
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5.
| Interleukin-4-dependent innate collaboration between iNKT cells and B-1 B cells controls adaptative contact sensitivity // Immunology, 2006. Vol. 117, N 4.-С.536-547
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6.
| Interleukin-4-dependent innate collaboration between iNKT cells and B-1 B cells controls adaptative contact sensitivity // Immunology, 2006. Vol. 117, N 4.-С.536-547
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7.
| TCR'альфа''бета',{+}CD8*{+} and TCR'гамма''дельта'{+} lymphocytes inhibit the capability of peritoneal macrophages to induce contact hypersensitivity // Folia biol.. -Polska, 2009. Vol. 57, N 1-2.-С.23-27
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8.
| Dermal dendritic cells, and not Langerhans cells, play an essential role in inducing an immune response // J. Immunol., 2008. Vol. 180, N 5.-С.3057-3064
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9.
| Essential roles of SHPS-1 in induction of contact hypersensitivity of skin // Immunol. Lett., 2008. Vol. 121, N 1.-С.52-60
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10.
| Fas-mediated inhibition of CD4{+} T cell priming results in dominance of type 1 CD8{+} T cells in the immune response to the contact sensitizer trinitrophenyl // J. Immunol., 2004. Vol. 173, N 5.-С.3178-3185
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11.
| Dendritic cells as a tool for the predictive identification of skin sensitisation hazard. The report and recommendations of ECVAM Workshop 51 Ispra, 7-8 Apr., 2004 // ATLA: Alternat. Lab. Anim., 2005. Vol. 33, N 1.-С.47-62
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12.
| Inducible ablation of mouse Langerhans cells diminishes but fails to abrogate contact hypersensitivity // J. Cell Biol., 2005. Vol. 169, N 4.-С.569-576
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13.
| Dermal mast cells determine susceptibility to ultraviolet B-induced systemic suppression of contact hypersensitivity responses in mice // J. Exp. Med., 1998. Vol. 187, N 12.-С.2045-2053
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14.
| Th2 cytokines, IgE and mast cells play a crucial role in the induction of para-phenylenediamine-induced contact hypersensitivity in mice // Clin. and Exp. Immunol., 2003. Vol. 132, N 3.-С.385-392
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15.
| Dendritic cells as a tool for the predictive identification of skin sensitisation hazard. The report and recommendations of ECVAM Workshop 51 Ispra, 7-8 Apr., 2004 // ATLA: Alternat. Lab. Anim., 2005. Vol. 33, N 1.-С.47-62
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16.
| van Iperen H.P. An animal model for extracorporeal photochemotherapy based on contact hypersensitivity // J. Photochem. and Photobiol. B, 1992. Vol. 15, N 4.-С.361-366
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17.
| Kodari Localand systemic-mediated suppression of contact hypersensitivity in mice by several structurally unrelated classes of tumor promoters // Carcinogenesis, 1991. Vol. 12, N 10.-С.1933-1937
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18.
| Контактната свръхчувствителност в напреднала възраст // Дерматол. и венерол., 1990. Vol. 29, N 4.-С.19-25
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19.
| Kodari E. Differential modulation of contact hypersensitivity and delayed hypersensitivity reactions by topical application of 12-О-tetradecanoylphorbol-13-acetate // Immunopharmacol. and Immunotoxicol., 1993. Vol. 15, N 5.-С.639-647
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20.
| A novel immunosuppressive compound // Int. J. immunopharmacol, 1991. Vol. 13, N 6.-С.805
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