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 1-20    21-40   41-55 
1.
Cigarette smoke exposure inhibits contact hypersensitivity via the generation of platelet-activating factor agonists // J. Immunol., 2013. Vol. 190, N 5.-С.2447-2454

2.
Cigarette smoke exposure inhibits contact hypersensitivity via the generation of platelet-activating factor agonists // J. Immunol., 2013. Vol. 190, N 5.-С.2447-2454

3.
ONZIN deficiency attenuates contact hypersensitivity responses in mice // Immunol. and Cell Biol., 2012. Vol. 90, N 7.-С.733-742

4.
IL-13 from T[h]2-type cells suppresses induction of antigen-specific T[h]1 immunity in a T-cell lymphoma // Int. Immunol., 2010. Vol. 22, N 1.-С.53-63

5.
TCR'альфа''бета',{+}CD8*{+} and TCR'гамма''дельта'{+} lymphocytes inhibit the capability of peritoneal macrophages to induce contact hypersensitivity // Folia biol.. -Polska, 2009. Vol. 57, N 1-2.-С.23-27

6.
Possible involvement of soluble B7-H4 in T cell-mediated inflammatory immune responses // Biochem. and Biophys. Res. Commun., 2009. Vol. 389, N 2.-С.349-353

7.
Dendritic cells from control but not atopic donors respond to contact and respiratory sensitizer treatment in vitro with differential cytokine production and altered stimulatory capacity // Clin. and Exp. Allergy, 2008. Vol. 38, N 7.-С.1148-1159

8.
Essential roles of SHPS-1 in induction of contact hypersensitivity of skin // Immunol. Lett., 2008. Vol. 121, N 1.-С.52-60

9.
Dermal dendritic cells, and not Langerhans cells, play an essential role in inducing an immune response // J. Immunol., 2008. Vol. 180, N 5.-С.3057-3064

10.
Interleukin-4-dependent innate collaboration between iNKT cells and B-1 B cells controls adaptative contact sensitivity // Immunology, 2006. Vol. 117, N 4.-С.536-547

11.
Interleukin-4-dependent innate collaboration between iNKT cells and B-1 B cells controls adaptative contact sensitivity // Immunology, 2006. Vol. 117, N 4.-С.536-547

12.
Inducible ablation of mouse Langerhans cells diminishes but fails to abrogate contact hypersensitivity // J. Cell Biol., 2005. Vol. 169, N 4.-С.569-576

13.
Dendritic cells as a tool for the predictive identification of skin sensitisation hazard. The report and recommendations of ECVAM Workshop 51 Ispra, 7-8 Apr., 2004 // ATLA: Alternat. Lab. Anim., 2005. Vol. 33, N 1.-С.47-62

14.
Dendritic cells as a tool for the predictive identification of skin sensitisation hazard. The report and recommendations of ECVAM Workshop 51 Ispra, 7-8 Apr., 2004 // ATLA: Alternat. Lab. Anim., 2005. Vol. 33, N 1.-С.47-62

15.
Fas-mediated inhibition of CD4{+} T cell priming results in dominance of type 1 CD8{+} T cells in the immune response to the contact sensitizer trinitrophenyl // J. Immunol., 2004. Vol. 173, N 5.-С.3178-3185

16.
Th2 cytokines, IgE and mast cells play a crucial role in the induction of para-phenylenediamine-induced contact hypersensitivity in mice // Clin. and Exp. Immunol., 2003. Vol. 132, N 3.-С.385-392

17.
IL-10 contributes to the inhibition of contact hypersensitivity in mice treated with photodynamic therapy // J. Immunol., 2000. Vol. 164, N 5.-С.2457-2462

18.
IL-10 contributes to the inhibition of contact hypersensitivity in mice treated with photodynamic therapy // J. Immunol., 2000. Vol. 164, N 5.-С.2457-2462

19.
IL-10 contributes to the inhibition of contact hypersensitivity in mice treated with photodynamic therapy // J. Immunol., 2000. Vol. 164, N 5.-С.2457-2462

20.
Cytotoxicity is mandatory for CD8{+} T cell mediated contact hypersensitivity // J. Exp. Med., 1999. Vol. 189, N 5.-С.779-786

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