Главная Назад


Авторизация
Идентификатор пользователя / читателя
Пароль (для удалённых пользователей)
 

Вид поиска

Область поиска
в найденном
Найдено в других БД
Формат представления найденных документов:
библиографическое описание краткийполный
Отсортировать найденные документы по:
авторузаглавиюгоду изданиятипу документа
Поисковый запрос: (<.>S=КЛЕТКИ FRTL-5<.>)
Общее количество найденных документов : 100
Показаны документы с 1 по 20
 1-20    21-40   41-60   61-80   81-100  
1.
Phorbol ester inhibits DNA synthesis induced by interleukin-6 in TSH-pretreated FRTL-5 cells // Endocrine Res., 1994. Vol. 20, N 2.-С.193-200

2.
Tornquist Thapsigargin-Induced calcium entry in FRTL-5 cells: Possible dependence on phospholipase A[2] activation // J. Cell. Physiol., 1994. Vol. 160, N 1.-С.40-46

3.
Inositol 1,4,5-trisphosphate 3-kinase activity in FRTL-5 cells: regulation of the enzyme activity by TSH // J. Endocrinol., 1995. Vol. 144, N 3.-С.527-532

4.
Genistein but not staurosporine can inhibit the mitogenic signal evoked by lithium in rat thyroid cells (FRTL-5) // J. Endocrinol., 1994. Vol. 143, N 2.-С.221-226

5.
Measurement of thyroid stimulating immunoglobulins in a new cell line transfected with a functional human TSH receptor (JPO9 cells), compared with an assay using FRTL-5 cells // Clin. Endocrinol., 1994. Vol. 40, N 5.-С.645-652

6.
Wang A phospholipase C inhibitor, U-73122, blocks TSH-induced inositol triphosphate production, Ca{2+} increase and arachidonic acid release in FRTL-5 thyroid cells // Biochim. et biophys. acta. Mol. Cell Res., 1994. Vol. 1223, N 1.-С.101-106

7.
Ekokoski Effects of 5,8,11,14-eicosatetraynoic acid on thapsigargin-induced calcium entry, and intracellular pH in thyroid FRTL-5 cells // Biochim. et biophys. acta. Mol. Cell Res., 1994. Vol. 1223, N 2.-С.274-278

8.
Wang A phospholipase C inhibitor, U-73122, blocks TSH-induced inositol triphosphate production, Ca{2+} increase and arachidonic acid release in FRTL-5 thyroid cells // Biochim. et biophys. acta. Mol. Cell Res., 1994. Vol. 1223, N 1.-С.101-106

9.
Shoda Graves' immunoglobulin G stimulates iodide efflux in FRTL-5 thyroid cells // J. Clin. Endocrinol. and Metab., 1993. Vol. 77, N 1.-С.94-97

10.
Epidermal growth factor stimulates cell proliferation and inhibits iodide uptake of FRTL-5 cells in vitro // J. Endocrinol., 1995. Vol. 145, N 3.-С.513-520

11.
Stimulation of Na{+}-K{+}-ATPase by thotropin in cultured thyroid follicular cells // Amer. J. Physiol., 1995. Vol. 268, N 5.-С.1252-1258

12.
Epidermal growth factor stimulates cell proliferation and inhibits iodide uptake of FRTL-5 cells in vitro // J. Endocrinol., 1995. Vol. 145, N 3.-С.513-520

13.
Tornquist Evidence for a pertussis toxin sensitive calcium entry pathway in thyroid FRTL-5 cells // J. Cell. Physiol., 1995. Vol. 164, N 1.-С.142-147

14.
Tornquist K. Inhibition of agonist-mediated calcium entry by calmodulin antagonists and by the Ca{2+}/calmodulin kinase II inhibitor KN-62. Studies with thyroid FRTL-5 cells // J. Endocrinol., 1996. Vol. 148, N 1.-С.131-138

15.
Tornquist K. Inhibition of agonist-mediated calcium entry by calmodulin antagonists and by the Ca{2+}/calmodulin kinase II inhibitor KN-62. Studies with thyroid FRTL-5 cells // J. Endocrinol., 1996. Vol. 148, N 1.-С.131-138

16.
Tornquist Evidence for a pertussis toxin sensitive calcium entry pathway in thyroid FRTL-5 cells // J. Cell. Physiol., 1995. Vol. 164, N 1.-С.142-147

17.
Kambe Thyrotropin and insulin increase the binding activity of thyroid-transcription factors in FRTL-5 cells // Environ. Med., 1995. Vol. 39, N 1.-С.25-28

18.
Tornquist Purinergic agonist ATP is a comitogen in thyroid FRTL-5 cells // J. Cell. Physiol., 1996. Vol. 166, N 2.-С.241-248

19.
Kambe Thyrotropin and insulin increase the binding activity of thyroid-transcription factors in FRTL-5 cells // Environ. Med., 1995. Vol. 39, N 1.-С.25-28

20.
Purinergic agonists stimulate the secretion of endothelin-1 in rat thyroid FRTL-5 cells // J. Cell. Physiol., 1996. Vol. 169, N 3.-С.538-5434

 1-20    21-40   41-60   61-80   81-100  
 




© Международная Ассоциация пользователей и разработчиков электронных библиотек и новых информационных технологий
(Ассоциация ЭБНИТ)