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Поисковый запрос: (<.>A=Wu, Chuanyue$<.>)
Общее количество найденных документов : 16
Показаны документы с 1 по 16
1.
Wu A novel role for 'альфа'3'бета'1 integrins in extracellular matrix assembly // J. Cell Sci., 1995. Vol. 108, N 6.-С.2511-2523

2.
Wu Integrin-linked kinase and PINCH: Partners in regulation of cell-extracellular matrix interaction and signal transduction // J. Cell Sci., 1999. Vol. 112, N 24.-С.4485-4489

3.
Li Integrin-linked kinase is localized to cell-matrix focal adhesions but not cell-cell adhesion sites and the focal adhesion localization of integrin-linked kinase is regulated by the PINCH-binding ANK repeats // J. Cell Sci., 1999. Vol. 112, N 24.-С.4589-4599

4.
Wu ILK interactions // J. Cell Sci., 2001. Vol. 114, N 14.-С.2549-2550

5.
A new focal adhesion protein that interacts with integrin-linked kinase and regulates cell adhesion and spreading // J. Cell Biol., 2001. Vol. 153, N 3.-С.585-598

6.
A critical role of the PINCH-integrin-linked kinase interaction in the regulation of cell shape change and migration // J. Biol. Chem., 2002. Vol. 277, N 1.-С.318-326

7.
Assembly of the PINCH-ILK-CH-ILKBP complex precedes and is essential for localization of each component to cell-matrix adhesion sites // J. Cell Sci., 2002. Vol. 115, N 24.-С.4777-4786

8.
EBV-expressing AGS gastric carcinoma cell sublines present increased motility and invasiveness // Int. J. Cancer, 2002. Vol. 99, N 5.-С.644-651

9.
EBV-expressing AGS gastric carcinoma cell sublines present increased motility and invasiveness // Int. J. Cancer, 2002. Vol. 99, N 5.-С.644-651

10.
EBV-expressing AGS gastric carcinoma cell sublines present increased motility and invasiveness // Int. J. Cancer, 2002. Vol. 99, N 5.-С.644-651

11.
PINCH-1 is an obligate partner of integrin-linked kinase (ILK) functioning in cell shape modulation, motility, and survival // J. Biol. Chem., 2003. Vol. 278, N 51.-С.51324-51333

12.
Migfilin and Mig-2 link focal adhesions to filamin and the actin cytoskeleton and function in cell shape modulation // Cell, 2003. Vol. 113, N 1.-С.37-47

13.
NO-dependent osteoclast motility: Reliance on cGMP-dependent protein kinase I and VASP // J. Cell Sci., 2005. Vol. 118, N 23.-С.5479-5487

14.
Wu Migfilin and its binding partners: From cell biology to human diseases // J. Cell Sci., 2005. Vol. 118, N 4.-С.659-664

15.
Migfilin's elimination from osteoarthritic chonrocytes further promotes the osteoarthritic phenotype via 'бета'-catenin upregulation // Biochem. and Biophys. Res. Commun., 2013. Vol. 430, N 2.-С.494-499

16.
Cancer Stem Cells Protect Non-Stem Cells From Anoikis: Bystander Effects // J. Cell. Biochem., 2016. Vol. 117, N 10.-С.2289-2301

 




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