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Общее количество найденных документов : 20
Показаны документы с 1 по 20
1.
Storey Mutation of the zing finger of HPV16 Е7 does not abolish CO-transformation with ras // J. Cell Biochem., 1989, Suppl. 13С.-С.215

2.
Storey Co-transformation by human papillomavirus types 6 and 11 // J. Gen. Virol., 1990. Vol. 71, N 1.-С.165-171

3.
Mutations of the human papillomavirus type 16 Е7 gene that affect transformation, transactivation and phosphorylation by the E7 protein // J. Gen. Virol., 1990. Vol. 71, N 4.-С.965-970

4.
Lack of immortolizing activity of a human papillomavirus type 16 variant DNA with a mutation in the E2 gene isolated from normal human cervical keratinocytes // Oncogene, 1992. Vol. 7, N 3.-С.459-465

5.
Characterization of the human papillomavirus E2 protein: Evidence of trans-activation and trans-repression in cervical keratinocytes // EMBO Journal, 1994. Vol. 13, N 22.-С.5451-5459

6.
The human papillomavirus type 16 E5 gene cooperates with the E7 gene to stimulate proliferation of primary cells and increases viral gene expression // Virology, 1994. Vol. 203, N 1.-С.73-80

7.
Mutations in the human papillomavirus type 16 E2 protein identify a region of the protein involved in binging to E1 protein // J. Gen. Virol., 1995. Vol. 76, N 4.-С.819-826

8.
Mutations in the human papillomavirus type 16 E2 protein identify multiple regions of the protein involved in binding to E1 // J. Gen. Virol., 1995. Vol. 76, N 11.-С.2909-2913

9.
Conditional immortalization of primary cells by human papillomavirus type 18 E6 and EJ-ras defines an E6 activity in G[0]/G[1] phase which can be substituted for mutations in p53 // Oncogene, 1995. Vol. 11, N 7.-С.653-661

10.
Conditional immortalization of primary cells by human papillomavirus type 18 E6 and EJ-ras defines an E6 activity in G[0]/G[1] phase which can be substituted for mutations in p53 // Oncogene, 1995. Vol. 11, N 7.-С.653-661

11.
Regulation of human papillomavirus type 16 DNA replication by E2, glucocorticoid hormone and epidermal growth factor // J. Gen. Virol., 1997. Vol. 78, N 8.-С.1963-1970

12.
A comparison study of gastric cancer risk in patients with duodenal and gastric ulcer: Roles of gastric mucosal histology and p53 codon 72 polymorhism // Dig. Diseases and Sci., 2004. Vol. 49, N 2.-С.254-259

13.
A comparison study of gastric cancer risk in patients with duodenal and gastric ulcer: Roles of gastric mucosal histology and p53 codon 72 polymorhism // Dig. Diseases and Sci., 2004. Vol. 49, N 2.-С.254-259

14.
A comparison study of gastric cancer risk in patients with duodenal and gastric ulcer: Roles of gastric mucosal histology and p53 codon 72 polymorhism // Dig. Diseases and Sci., 2004. Vol. 49, N 2.-С.254-259

15.
A comparison study of gastric cancer risk in patients with duodenal and gastric ulcer: Roles of gastric mucosal histology and p53 codon 72 polymorhism // Dig. Diseases and Sci., 2004. Vol. 49, N 2.-С.254-259

16.
Akgul HPV-associated skin disease // J. Pathol., 2006. Vol. 208, N 2.-С.165-175

17.
Akgul HPV-associated skin disease // J. Pathol., 2006. Vol. 208, N 2.-С.165-175

18.
Akgul HPV-associated skin disease // J. Pathol., 2006. Vol. 208, N 2.-С.165-175

19.
Fox BMX negatively regulates BAK function increasing apoptotic resistance to chemotherapeutic agents in cancer cells // Anticancer Res., 2014. Vol. 34, N 10.-С.6188

20.
Fox BMX negatively regulates BAK function increasing apoptotic resistance to chemotherapeutic agents in cancer cells // Anticancer Res., 2014. Vol. 34, N 10.-С.6188

 




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