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1.
Characterization o T-cell receptor 'альфа''бета' repertoire in synovial tissue from different temporal phases of rheumatoid arthritis // 11th Eur. Immunol. Meet., Espoo, 9-12 June, 1991. -Helsinki, 1991.-С.35

2.
Zamvil Scott S. The T lymphocyte in experimental allergic encephalomyelitis // Annu. Rev. Immunol.. -Palo Alto (Calif.), 1990, Vol. 8.-С.579-621

3.
Steinman The discovery of natalizumab, a potent therapeutic for multiple sclerosis // J. Cell Biol., 2012. Vol. 199, N 3.-С.413-416

4.
Optimization of current and future therapy for autoimmune diseases // Nature Med., 2012. Vol. 18, N 1.-С.59-65

5.
Tyrosine kinase inhibitors ameliorate autoimmune encephalomyelitis in a mouse model of multiple sclerosis // J. Clin. Immunol., 2011. Vol. 31, N 6.-С.1010-1020

6.
Involvement of distinct murine T-cell receptors in the autoimmune encephalitogenic response to nested epitopes of myelin basic protein // Proc. Nat. Acad. Sci. USA, 1988. Vol. 85, N 22.-С.8608-8612

7.
Involvement of distinct murine T-cell receptors in the autoimmune encephalitogenic response to nested epitopes of myelin basic protein // Proc. Nat. Acad. Sci. USA, 1988. Vol. 85, N 22.-С.8608-8612

8.
Limited heterogeneity of T cell receptors from lymphocytes mediating autoimmune encephalomyelitis allows specific immune intervention // Cell, 1988. Vol. 54, N 2.-С.263-273

9.
A molecular basis for MHC class II - associated autoimmunity // Science, 1988. Vol. 240, N 4855.-С.1003-1009

10.
The absence of preferential T cell receptor V'бета' gene usage at the site of demyelination in experimental allergic encephalomyelitis (EAE) // Austral. and N. Z. J. Med., 1992. Vol. 22, N 4.-С.420

11.
Regulation of different inflammatory diseases by impacting the mevalonate pathway // Immunology, 2009. Vol. 127, N 2.-С.18-25

12.
Treatment of autoimmune neuroinflammation with a synthetic tryptophan metabolite // Science, 2005. Vol. 310, N 5749.-С.850-855

13.
A key regulatory role for histamine in experimental autoimmune encephalomyelitis: Disease exacerbation in histidine decarboxylase-deficient mice // J. Immunol., 2006. Vol. 176, N 1.-С.17-26

14.
HMG-CoA reductase inhibition causes increased necrosis and apoptosis in an in vivo mouse glioblastoma multiforme model // Anticancer Res., 2009. Vol. 29, N 12.-С.4901-4908

15.
HMG-CoA reductase inhibition causes increased necrosis and apoptosis in an in vivo mouse glioblastoma multiforme model // Anticancer Res., 2009. Vol. 29, N 12.-С.4901-4908

16.
Differential regulation of chemokines by IL-17 in colonic epithelial cells // J. Immunol., 2008. Vol. 181, N 9.-С.6536-6545

17.
HMG-CoA reductase inhibition causes increased necrosis and apoptosis in an in vivo mouse glioblastoma multiforme model // Anticancer Res., 2009. Vol. 29, N 12.-С.4901-4908

18.
HLA-DP'бета' and susceptibility to multiple sclerosis: An analysis of caucasoid and japanese patient populations // Hum. Immunol., 1990. Vol. 28, N 4.-С.365-72

19.
Steinman Development of antigen-specific therapies for autoimmune disease // Mol. Biol. and Med., 1990. Vol. 7, N 4.-С.333-339

20.
Predominant expression of T cell receptor V7 in tumor-infiltrating lymphocytes of uveal melanoma // Science, 1990. Vol. 249, N 4969.-С.672-674

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