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Общее количество найденных документов : 38
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 1-20    21-38 
1.
Sjolin Calcium-induced translocation of annexins to subcellular organelles of human neutrophils // Biochem. J., 1994. Vol. 300, N 2.-С.325-330

2.
Histaminergic regulation of NK cells: Role of monocyte-derived reactive oxygen metabolites // J. Immunol., 1994. Vol. 153, N 11.-С.4940-4947

3.
Mobilization of granules and secretory vesicles during in vivo exudation of human neutrophils // J. Immunol., 1995. Vol. 154, N 8.-С.4157-4165

4.
Histaminergic regulation of NK cells: Role of monocyte-derived reactive oxygen metabolites // J. Immunol., 1994. Vol. 153, N 11.-С.4940-4947

5.
Mobilization of granules and secretory vesicles during in vivo exudation of human neutrophils // J. Immunol., 1995. Vol. 154, N 8.-С.4157-4165

6.
Different subcellular localization of cytochrome b and the dormant NADPH-oxidase in neutrophils and macrophages: Effect on the production of reactive oxygen species during phagocytosis // Cell. Immunol., 1995. Vol. 161, N 1.-С.61-71

7.
Chemoattractant-induced NADPH oxidase activity in human monocytes is terminated without any association of receptor-ligand complex to cytoskeleton // Inflammation, 1995. Vol. 19, N 2.-С.179-191

8.
A sensitive isoluminol-enhanced chemiluminescence technique to measure release of reactive oxygen species in human neutrophils // Eur. J. Clin. Invest., 1995. Vol. 25, Suppl. n 2.-С.26

9.
A simple chemiluminescence assay for the determination of reactive oxygen species produced by human neutrophils // J. Immunol. Meth., 1996. Vol. 192, N 1-2.-С.173-178

10.
A simple chemiluminescence assay for the determination of reactive oxygen species produced by human neutrophils // J. Immunol. Meth., 1996. Vol. 192, N 1-2.-С.173-178

11.
A rise in ionizes calcium activates the neutrophil NADPH-oxidase but is not sufficient to directly translocate cytosolic p47[phox] or p67[phox] to b cytochrome containing membranes // Inflammation, 1997. Vol. 21, N 5.-С.531-540

12.
Desensitization of formyl peptide receptors is abolished in calcium ionophore-primed neutrophils: An association of the ligand-receptor complex to the cytoskeleton is not required for a rapid termination of the NADPH-oxidase response // J. Immunol., 1998. Vol. 160, N 5.-С.2463-2468

13.
Desensitization of formyl peptide receptors is abolished in calcium ionophore-primed neutrophils: An association of the ligand-receptor complex to the cytoskeleton is not required for a rapid termination of the NADPH-oxidase response // J. Immunol., 1998. Vol. 160, N 5.-С.2463-2468

14.
Serotonin protects natural killer-cells from inhibition induced by monocyte-derived reactive oxygen metabolites // Eur. J. Clin. Invest., 1999. Vol. 29, Suppl. 1.-С.44

15.
Lipopolysaccharide-induced mobilization of neutrophil granules primes the cells to galectin-3 // Eur. J. Clin. Invest., 1999. Vol. 29, Suppl. 1.-С.45

16.
Serotonin protects natural killer-cells from inhibition induced by monocyte-derived reactive oxygen metabolites // Eur. J. Clin. Invest., 1999. Vol. 29, Suppl. 1.-С.44

17.
Galectin-3 activates the NADPH-oxidase in exudated but not peripheral blood neutrophils // Blood, 1998. Vol. 91, N 9.-С.3430-3438

18.
The phagocyte chemiluminescence paradox: Luminol can act as an inhibitor of neutrophil NADPH-oxidase activity // Luminescence, 1999. Vol. 14, N 3.-С.153-160

19.
Identification of CD66a and CD66b as the major galectin-3 receptor candidates in human neutrophils // J. Immunol., 1999. Vol. 163, N 10.-С.5592-5598

20.
Dahlgren Respiratory burst in human neutrophils // J. Immunol. Meth., 1999. Vol. 232, N 1-2.-С.3-14

 1-20    21-38 
 




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