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 1-20    21-38 
1.
The host defence peptide LL-37 selectively permeabilizes apoptotic leukocytes // Antimicrob. Agents and Chemother., 2009. Vol. 53, N 3.-С.1027-1038

2.
Tumour necrosis factor (TNF)-'альфа' primes murine neutrophils when triggered via formyl peptide receptor-related sequence 2, the murine orthologue of human formyl peptide receptor-like 1, through a process involving the type I TNF receptor and subcellular granule mobilization // Immunology, 2008. Vol. 125, N 4.-С.591-600

3.
The host defence peptide LL-37 selectively permeabilizes apoptotic leukocytes // Antimicrob. Agents and Chemother., 2009. Vol. 53, N 3.-С.1027-1038

4.
The host defence peptide LL-37 selectively permeabilizes apoptotic leukocytes // Antimicrob. Agents and Chemother., 2009. Vol. 53, N 3.-С.1027-1038

5.
Pellme The two neutrophil plasma membrane markers alkaline phosphatase and HLA class I antigen localize differently in granule-deficient cytoplasts. An ideal plasma membrane marker in human neutrophils is still lacking // J. Immunol. Meth., 2007. Vol. 325, N 1-2.-С.88-95

6.
Svensson Respiratory burst induced in human eosinophilic granulocytes by shigella // Scand. J. Immunol., 2001. Vol. 54, прил. 1.-С.T82

7.
Dahlgren Respiratory burst in human neutrophils // J. Immunol. Meth., 1999. Vol. 232, N 1-2.-С.3-14

8.
Ability of monocyte-derived dendritic cells to secrete oxygen radicals in response to formyl peptide receptor family agonists compared to that of myeloid and plasmacytoid dendritic cells // Clin. and Vaccine Immunol., 2007. Vol. 14, N 3.-С.328-330

9.
Changes in activation states of murine polymorphonuclear leukocytes (PMN) during inflammation: A comparison of bone marrow and peritoneal exudate PMN // Clin. and Vaccine Immunol., 2006. Vol. 13, N 5.-С.575-583

10.
Dahlgren Effects on extra- and intracellularly localized, chemoattractant-induced, oxygen radical production in neutrophils following modulation of conditions for ligand-receptor interaction // Inflammation, 1988. Vol. 12, N 4.-С.335-349

11.
Dahlgren Degranulation in human neutrophils primes the cells for subsequent responsiveness to the chemoattractant N-formylmethyonylleucylphenylalanine but does not increase the sensitivity of the NADPH-oxidase to an intracellular calcium rise // Biochim. et biophys. acta. Mol. Cell. Res., 1990. Vol. 1052, N 1.-С.42-46

12.
Follin Altered O[2]~H[2]O[2] production ratio by in vitro and in vivo primed human neutrophils // Biochem. and Biophys. Res. Commun., 1990. Vol. 167, N 3.-С.970-976

13.
Activation of the oxygen-radical-generating system in granules of intact human neutrophils by a calcium ionophore (ionomycin) // Biochim. et biophys. acta. Mol. Cell Res., 1992. Vol. 1137, N 2.-С.182-188

14.
Follin Phagocytosis by lipopolysaccharideprimed human neutrophils is associated with increased extracellular release of reactive oxygen metabolites // Inflammation, 1992. Vol. 16, N 2.-С.83-91

15.
Neutrophil killing of two type 1 fimbria-bearing Escherichia coli strains: Dependence on respiratory burst activation // Infec. and immun., 1990. Vol. 58, N 1.-С.37-42

16.
Dahlgren Difference in hydrogen peroxide release between human neutrophils and neutrophil cytoplasts following calcium ionophore activation. A role of the subcellular granule in activation of the NADPH-oxidase in human neutrophils? // Biochim. et biophys. acta. Mol. Cell. Res., 1989. Vol. 1010, N 1.-С.41-48

17.
Dahlgren The limitation of the human neutrophil chemiluminescence response by extracellular peroxidase is stimulus dependent: effect of added horse radish peroxidase on the response induced by both soluble and particulate stimuli // J. Clin. and Lab. Immunol., 1988. Vol. 26, N 1.-С.49-53

18.
Cohansson Luminol-arnplified chemiluminescence activity in human monocytes: A comparison ith the activity induced in granulocytes // J. Bioluminescence and Chemiluminescence, 1990. Vol. 5, N 1.-С.37-41

19.
Human neutrophil chemiluminescence and f-Meth-Leu-Phe receptor exposure in bacterial infections // APMIS, 1989. Vol. 97, N 7.-С.585-590

20.
Chemoattractant-induced NADPH oxidase activity in human monocytes is terminated without any association of receptor-ligand complex to cytoskeleton // Inflammation, 1995. Vol. 19, N 2.-С.179-191

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